检索范围:
排序: 展示方式:
Yingying Zhao, Mengfan Wu, Zhiyong Ji, Yuanyuan Wang, Jiale Li, Jianlu Liu, Junsheng Yuan
《化学科学与工程前沿(英文)》 2019年 第13卷 第4期 页码 832-844 doi: 10.1007/s11705-019-1871-7
关键词: mineral carbonation wet SO2 disposal seawater desulfurization
Performance and mechanism of carbamazepine removal by FeS-SO process: experimental investigation and
《环境科学与工程前沿(英文)》 2023年 第17卷 第9期 doi: 10.1007/s11783-023-1713-1
● Synergistic removal of carbamazepine (CBZ) was obtained in the FeS-S2O82– process.
关键词: FeS S2O82– Carbamazepine DFT calculations Degradation routes
Preparation and characterization of SO42-/TiO2 and S2O82-/TiO2 catalysts
MA Xuedan, GUO Daishi, JIANG Qizhong, MA Zifeng, MA Zhengfei, YE Weidong, LI Chunbo
《化学科学与工程前沿(英文)》 2007年 第1卷 第1期 页码 45-49 doi: 10.1007/s11705-007-0009-5
关键词: exhibit Different persulfate adsorption-desorption catalytic
Minhua Zhang, Baojuan Huang, Haoxi Jiang, Yifei Chen
《化学科学与工程前沿(英文)》 2017年 第11卷 第4期 页码 594-602 doi: 10.1007/s11705-017-1668-5
关键词: metal-organic framework selective catalytic reduction manganese oxides deNOx SO2 resistance
《环境科学与工程前沿(英文)》 2022年 第16卷 第8期 doi: 10.1007/s11783-022-1529-4
• The resistance of phage PhiX174 to nZVI was much stronger than that of MS2.
关键词: Nanoscale zero-valent iron (nZVI) MS2 PhiΧ174 Resistance Inactivation Pathogenic microorganisms
赵君科,王保健,任先文,朱祖良
《中国工程科学》 2002年 第4卷 第2期 页码 74-78
利用建造在四川绵阳科学城热电厂的最大烟气处理量为20000 m3/h的脉冲电晕等离子体烟气脱硫工业中试装置,研究了影响脱硫效率的因素,获得了最佳工艺参数。究结果表明,当烟气温度在65~70℃,烟气水分含量约10%,氨硫化学剂量比为1∶1,烟停留时间大于6 s、能耗低于5 Wh/Nm3等条件下,脱硫率达85%以上。
Review of SO42-/MxOy solid superacid catalysts
Yanni WU, Shijun LIAO
《化学科学与工程前沿(英文)》 2009年 第3卷 第3期 页码 330-343 doi: 10.1007/s11705-009-0208-3
关键词: solid superacid catalyst synthesis method characterization applications
Ruizhuang ZHAO, Ju SHANGGUAN, Yanru LOU, Jin SONG, Jie MI, Huiling FAN
《化学科学与工程前沿(英文)》 2010年 第4卷 第4期 页码 423-428 doi: 10.1007/s11705-010-0503-z
关键词: high-temperature coal gas Fe2O3 desulfurization sorbent SO2 atmosphere regeneration behaviors sulfur recovery
Mechanism of insulin resistance in obesity: a role of ATP
《医学前沿(英文)》 2021年 第15卷 第3期 页码 372-382 doi: 10.1007/s11684-021-0862-5
关键词: type 2 diabetes energy expenditure mitochondria hyperinsulinemia hyperglucagonemia AMPK
Xiaolei LI, Chunying ZHU, Youguang MA
《化学科学与工程前沿(英文)》 2013年 第7卷 第2期 页码 185-191 doi: 10.1007/s11705-013-1326-5
关键词: SO2 removal bubble column reactor removal rate ammonium bicarbonate absorbent
Mechanisms of insulin resistance in obesity
null
《医学前沿(英文)》 2013年 第7卷 第1期 页码 14-24 doi: 10.1007/s11684-013-0262-6
Obesity increases the risk for type 2 diabetes through induction of insulin resistance. Treatment of type 2 diabetes has been limited by little translational knowledge of insulin resistance although there have been several well-documented hypotheses for insulin resistance. In those hypotheses, inflammation, mitochondrial dysfunction, hyperinsulinemia and lipotoxicity have been the major concepts and have received a lot of attention. Oxidative stress, endoplasmic reticulum (ER) stress, genetic background, aging, fatty liver, hypoxia and lipodystrophy are active subjects in the study of these concepts. However, none of those concepts or views has led to an effective therapy for type 2 diabetes. The reason is that, there has been no consensus for a unifying mechanism of insulin resistance. In this review article, literature is critically analyzed and reinterpreted for a new energy-based concept of insulin resistance, in which insulin resistance is a result of energy surplus in cells. The energy surplus signal is mediated by ATP and sensed by adenosine monophosphate-activated protein kinase (AMPK) signaling pathway. Decreasing ATP level by suppression of production or stimulation of utilization is a promising approach in the treatment of insulin resistance. In support, many of existing insulin sensitizing medicines inhibit ATP production in mitochondria. The effective therapies such as weight loss, exercise, and caloric restriction all reduce ATP in insulin sensitive cells. This new concept provides a unifying cellular and molecular mechanism of insulin resistance in obesity, which may apply to insulin resistance in aging and lipodystrophy.
关键词: type 2 diabetes energy expenditure inflammation lipotoxicity mitochondria hyperinsulinemia adenosine monophosphate-activated protein kinase (AMPK)
Effective regeneration of thermally deactivated commercial V-W-Ti catalysts
Xuesong SHANG, Jianrong LI, Xiaowei YU, Jinsheng CHEN, Chi HE
《化学科学与工程前沿(英文)》 2012年 第6卷 第1期 页码 38-46 doi: 10.1007/s11705-011-1167-z
关键词: V2O5-WO3/TiO2 catalysts thermal deactivation regeneration NH4Cl dilute H2SO4 solution
Wenjie Zhu, Binghe Xu
《医学前沿(英文)》 2021年 第15卷 第2期 页码 208-220 doi: 10.1007/s11684-020-0795-4
关键词: endocrine-resistant HR+/HER2- advanced breast cancer randomized clinical trials meta-analysis targeted therapy
null
《医学前沿(英文)》 2015年 第9卷 第2期 页码 139-145 doi: 10.1007/s11684-015-0377-z
In obesity, chronic inflammation is believed to induce insulin resistance and impairs adipose tissue function. Although this view is supported by a large body of literature, it has been challenged by growing evidence that pro-inflammatory cytokines may favor insulin sensitivity through induction of energy expenditure. In this review article, interleukin 15 (IL-15) is used as a new example to explain the beneficial effects of the pro-inflammatory cytokines. IL-15 is secreted by multiple types of cells including macrophages, neutrophils and skeletal muscle cells. IL-15 expression is induced in immune cells by endotoxin and in muscle cells by physical exercise. Its transcription is induced by transcription factor NF-κB. IL-15 binds to its receptor that contains three different subunits (α, β and γ) to activate JAK/STAT, PI3K/Akt, IKK/NF-κB and JNK/AP1 pathways in cells. In the regulation of metabolism, IL-15 reduces weight gain without inhibiting food intake in rodents. IL-15 suppresses lipogenesis, stimulates brown fat function, improves insulin sensitivity through weight loss and energy expenditure. In human, circulating IL-15 is negatively associated with body weight. In the immune system, IL-15 stimulates proliferation and differentiation of T cells, NK cells, monocytes and neutrophils. In the anti-obesity effects of IL-15, T cells and NK cells are not required, but leptin receptor is required. In summary, evidence from human and rodents supports that the pro-inflammatory cytokine IL-15 may enhance energy expenditure to protect the body from obesity and type 2 diabetes. The mechanism of IL-15 action remains to be fully uncovered in the regulation of energy expenditure.
关键词: inflammation obesity cytokine energy expenditure insulin resistance
标题 作者 时间 类型 操作
A combination process of mineral carbonation with SO2 disposal for simulated flue gas by magnesia-added
Yingying Zhao, Mengfan Wu, Zhiyong Ji, Yuanyuan Wang, Jiale Li, Jianlu Liu, Junsheng Yuan
期刊论文
Performance and mechanism of carbamazepine removal by FeS-SO process: experimental investigation and
期刊论文
Preparation and characterization of SO42-/TiO2 and S2O82-/TiO2 catalysts
MA Xuedan, GUO Daishi, JIANG Qizhong, MA Zifeng, MA Zhengfei, YE Weidong, LI Chunbo
期刊论文
Metal-organic framework loaded manganese oxides as efficient catalysts for low-temperature selective catalytic reduction of NO with NH
Minhua Zhang, Baojuan Huang, Haoxi Jiang, Yifei Chen
期刊论文
The inactivation of bacteriophages MS2 and PhiX174 by nanoscale zero-valent iron: Resistance difference
期刊论文
Effective regeneration of thermally deactivated commercial V-W-Ti catalysts
Xuesong SHANG, Jianrong LI, Xiaowei YU, Jinsheng CHEN, Chi HE
期刊论文
Overcoming resistance to endocrine therapy in hormone receptor-positive human epidermal growth factorreceptor 2-negative (HR
Wenjie Zhu, Binghe Xu
期刊论文